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<article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xmlns:ali="http://www.niso.org/schemas/ali/1.0/" article-type="research-article" dtd-version="1.2" xml:lang="en"><front><journal-meta><journal-id journal-id-type="publisher-id">Kazan medical journal</journal-id><journal-title-group><journal-title xml:lang="en">Kazan medical journal</journal-title><trans-title-group xml:lang="ru"><trans-title>Казанский медицинский журнал</trans-title></trans-title-group></journal-title-group><issn publication-format="print">0368-4814</issn><issn publication-format="electronic">2587-9359</issn><publisher><publisher-name xml:lang="en">Eco-Vector</publisher-name></publisher></journal-meta><article-meta><article-id pub-id-type="publisher-id">83634</article-id><article-id pub-id-type="doi">10.17816/kazmj83634</article-id><article-categories><subj-group subj-group-type="toc-heading" xml:lang="en"><subject>Articles</subject></subj-group><subj-group subj-group-type="toc-heading" xml:lang="ru"><subject>Статьи</subject></subj-group><subj-group subj-group-type="article-type"><subject>Research Article</subject></subj-group></article-categories><title-group><article-title xml:lang="en">Target organs in arterial hypertension</article-title><trans-title-group xml:lang="ru"><trans-title>Органы-мишени при артериальной гипертонии</trans-title></trans-title-group></title-group><contrib-group><contrib contrib-type="author"><name-alternatives><name xml:lang="en"><surname>Galyavich</surname><given-names>A. S.</given-names></name><name xml:lang="ru"><surname>Галявич</surname><given-names>А. С.</given-names></name></name-alternatives><address><country country="RU">Russian Federation</country></address><email>info@eco-vector.com</email><xref ref-type="aff" rid="aff1"/></contrib></contrib-group><aff-alternatives id="aff1"><aff><institution xml:lang="en">Kazan State Medical University</institution></aff><aff><institution xml:lang="ru">Казанский государственный медицинский университет</institution></aff></aff-alternatives><pub-date date-type="pub" iso-8601-date="1997-10-15" publication-format="electronic"><day>15</day><month>10</month><year>1997</year></pub-date><volume>78</volume><issue>5</issue><issue-title xml:lang="en"/><issue-title xml:lang="ru"/><fpage>378</fpage><lpage>381</lpage><history><date date-type="received" iso-8601-date="2021-10-23"><day>23</day><month>10</month><year>2021</year></date><date date-type="accepted" iso-8601-date="2021-10-23"><day>23</day><month>10</month><year>2021</year></date></history><permissions><copyright-statement xml:lang="en">Copyright ©; 1997, Eco-Vector</copyright-statement><copyright-statement xml:lang="ru">Copyright ©; 1997, Эко-Вектор</copyright-statement><copyright-year>1997</copyright-year><copyright-holder xml:lang="en">Eco-Vector</copyright-holder><copyright-holder xml:lang="ru">Эко-Вектор</copyright-holder><ali:free_to_read xmlns:ali="http://www.niso.org/schemas/ali/1.0/"/></permissions><self-uri xlink:href="https://kazanmedjournal.ru/kazanmedj/article/view/83634">https://kazanmedjournal.ru/kazanmedj/article/view/83634</self-uri><abstract xml:lang="en"><p>One of the consequences of long-term BP increase is damage of internal organs, the so-called target organs (target organ damage, end organ damage). These include heart, brain, kidneys, vessels. Heart damage in arterial hypertension may be manifested by left ventricular hypertrophy, angina pectoris, myocardial infarction, chronic heart failure and sudden death, brain damage - by thrombosis and hemorrhages, hypertensive encephalopathy and cerebral lacunae, kidneys - microalbuminuria, proteinuria, chronic renal failure, vascular involvement - lesions of retinal vessels, carotid arteries, aorta (aneurysm).</p></abstract><trans-abstract xml:lang="ru"><p>Одним из следствий длительного повышения АД является поражение внутренних органов, так называемых органов-мишеней (target organ damage, end organ damage). К ним относятся сердце, мозг, почки, сосуды. Поражение при артериальной гипертонии сердца может проявляться гипертрофией левого желудочка, стенокардией, инфарктом миокарда, хронической сердечной недостаточностью и внезапной смертью, мозга — тромбозами и геморрагиями, гипертонической энцефалопатией и церебральными лакунами, почек — микроальбуминурией, протеинурией, хронической почечной недостаточностью, вовлеченность сосудов — поражением сосудов сетчатки глаз, сонных артерий, аорты (аневризма).</p></trans-abstract><kwd-group xml:lang="en"><kwd>Kazan Medical archive</kwd></kwd-group><kwd-group xml:lang="ru"><kwd>Казанский медицинский архив</kwd></kwd-group><funding-group/></article-meta></front><body></body><back><ref-list><ref id="B1"><label>1.</label><mixed-citation>Алмазов В. А., Шляхто Е.В., Соколова Л.А. Пограничная артериальная гипертензия. — СПб, 1992.</mixed-citation></ref><ref id="B2"><label>2.</label><mixed-citation>Aalkjaer С., Heagerty А.М. et al.// Cirс. 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